题目内容
（请给出正确答案）
[多选题]
Kahn以为儒家文化四种特质是（)。
A.家庭中社会化方式促成个体沉着节制，注重工作、家庭与责任
B.具备团队协作倾向
C.阶层意识，以为级别存在理所固然
D.人际关系具备互补性
E.具备明显个人主义倾向
查看答案
如果结果不匹配，请 联系老师 获取答案
题目内容
（请给出正确答案）
如果结果不匹配，请 联系老师 获取答案
题目内容
（请给出正确答案）
A.家庭中社会化方式促成个体沉着节制，注重工作、家庭与责任
B.具备团队协作倾向
C.阶层意识，以为级别存在理所固然
D.人际关系具备互补性
E.具备明显个人主义倾向
如果结果不匹配，请 联系老师 获取答案
更多“Kahn以为儒家文化四种特质是()。”相关的问题
A.学者们认为正常特质和异常特质是连续的
B.复杂的现实生活经历会使人格特质发生某些改变
C.“人心不同，各如其面”反映了人格的独特性与共同性
D.异常人格特质也有稳定的结构，且与正常人格特质没有关系
E.研究发现有四种异常人格特质，即“反社会”、“神经质”、“情绪失调”和“强迫”
A.积极的自我观念，接受自己，悦纳自己
B.恰当的认同他人，认可别人的存在和重要性
C.面对和接受现实，能设身处地、实事求是的去面对和接受现实的考验
D.主观经验丰富，可供取用
A．Y
B．N
C．NG
听力原文：W: Didn't you write a paper about Albert Kahn last semester？
M: Yes.for my history of architecture class.
W: Oh,I am taking it now andI have to do some research on industrial architecture.I need to read up on Kahn's factories. So I would like to see what you wrote about them.
M: I don't think my paper will help；I focus on his classical design like Clements library and office buildings. but you are interested in the modem building he is famous for.
W: Yes,he is best known for his factory,especially the auto plants in Detroit.He made a breakthrough in industrial design.You know before his time,factories were so narrow and inefficient,but his factory provided enough light and air and open space. So the cars could be assembled in one huge plant.
M: I remember reading that previously.factories had wooden frames and the heavy machinery made the buildings vibrate and there were fire hazards too.But when Kahn started to design auto plants around the turn of the century,reinforced concrete had just been invented.With the reinforced concrete,not only were the buildings solid and fireproof but they were cheap to put up too!
W: You seem to know a lot of about his industrial career.
M: Actually even though I wrote about his other works,I did a lot of background reading.Let me see if I can dig up that paper for you.There were some books of articles included in my bibliography that you might want to look up at the library.
W: Thank you.It will be a great help for me.
(20)
A．She needs to do some research on industrial architecture.
B．Albert Kahn is the woman's most-respected architect.
C．She is interested in his classical design and industrial design.
D．Albert Kahn designed many grand factories all over the world.
What do Irving Kahn and Ruth Bader Ginsburg have in common？
A.Neither of them is subject to forced retirement.
B.Neither of them desires reward for their work.
C.Both cling to their positions despite opposition
D.Both are capable of coping with heavy workloads.
Eat More, Weigh Less, Live Longer
Clever genetic detective work may have found out the reason why a near-starvation diet prolongs the life of many animals.
Ronald Kahn at Harvard Medical School in Boston, US, and his colleagues have been able to extend the lifespan （寿命） of mice by 18 per cent by blocking the rodent's （啮齿动物） increase of fat in specific cells. This suggests that thinness--and not necessarily diet--promotes long life in "calorie （热量卡） restricted" animals.
"It's very cool work," says aging researcher Cynthia Kenyon of the University of California, San Francisco. "These mice eat all they want, lose weight and live longer. It's like heaven."
Calorie restriction dramatically extends the lifespan of organisms as different as worms and rodents. Whether this works in humans is still unknown, partly because few people are willing to submit to such a strict diet.
But many researchers hope they will be able to trigger the same effect with a drug once they understand how less food leads to a longer life. One theory is that eating less reduces the increase of harmful things that can damage cells. But Kahn's team wondered whether the animals simply benefit by becoming thin.
To find out, they used biology tricks to disrupt the insulin （胰岛素） receptor （受体） gene in lab mice--but only in their fat cells. "Since insulin is needed to help fat cells store fat, these animals were protected against becoming fat," explains Kahn.
This slight genetic change in a single tissue had dramatic effects. By three months of age, Kahn's modified mice had up to 70 per cent less body fat than normal control mice, despite the fact that they ate 55 per cent more food per gram of body weight.
In addition, their lifespan increased. The average control mouse lived 753 days, while the thin rodents averaged a lifespan of 887 days. After three years, all the control mice had died, but one-quarter of the modified rodents were still alive.
"That they get these effects by just manipulating the fat cells is controversial," says Leonard Guarente of the Massachusetts Institute of Technology, who studies calorie restriction and aging.
But Guarente says Kahn has yet to prove that the same effect is responsible for increased lifespan in calorie-restricted animals. "It might be the same effect or there might be two routes to long life," he points out, "and that would be very interesting."
第 41 题 Ronald Kahn and his colleagues can make mice live longer by.
A．offering them less food.
B．giving them a balanced diet.
C．disrupting the specific genes in their fat cells.
D．preventing them growing larger.
A.ArpaNet协议
B.NSFNet协议
C.TCP/IP协议
D.OSI参考模型协议
根据短文回答 41～45 题。
Eat More, weigh Less, Live Longer
Clever genetic detective work may have found out the reason why a near-starvation diet prolongs the life of many animals.
"It's very cool work，"says aging researcher Cynthia Kenyon of the University of California，San Francisc0."These mice eat all they want.10se weight and live longer.It's Like heaven."
Calorie restriction dramatically extends the lifespan of organisms as different as worms and rodents.Whether this works in humans is still unknown，partly because few people are willing to submit to such a strict diet
But many researchers hope they will be able to trigger the same effect with a drug once they understand how less food leads to a longer life.One theory is that eating less reduces the increase of harmful things that can damage cells.But Kahn's team wondered whether the animals simply benefit by becoming thin。
To find out，they used biology tricks to disrupt the insulin(胰岛素)receptor(受体)gene in lab mice—but only in their fat cells."Since insulin is needed to help fat cells store fat，these animals were protected against becoming fat，"explains Kahn.
This slight genetic change in a single tissue had dramatic effects.By three months of age，Kahn's modified mice had up t0 70 per cent less body fat than normal control mice，despite the fact that they ate 55 per cent more food per gram of body weight.
In addition，their lifespan increased.The average control mouse lived 753 days，while the thin rodents averaged a lifespan of 887 days.After three years，all the control mice had died，but one—quarter of the modified rodents were still alive.
"That they get these effects by just manipulating the fat cells is controversial，"says Leonard Guarente of the Massachusetts Institute of Technology, who studies calorie restriction and agin9.
But Guarente says Kahn has yet to prove that the same effect is responsible for increased lifespan in calorie-restricted anima.1s."It might be the same effect or there might be two routes to long life，"he points out，"and that would be very interesting."
第 41 题 Ronald Kahn and his colleagues can make mice live longer by（）.
A．offering them less food.
B．giving them a balanced diet.
C．disrupting the specific genes in their fat cells.
D．preventing them growing larger.
